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Endotoxin of Porphyromonas gingivalis amplifies the inflammatory response in hyperglycemia-induced zebrafish through a mechanism involving chitinase-like protein YKL-40 analogs.
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G. Gündüz Et Al. , "Endotoxin of Porphyromonas gingivalis amplifies the inflammatory response in hyperglycemia-induced zebrafish through a mechanism involving chitinase-like protein YKL-40 analogs.," Toxicological research , vol.39, no.4, pp.625-636, 2023

Gündüz, G. Et Al. 2023. Endotoxin of Porphyromonas gingivalis amplifies the inflammatory response in hyperglycemia-induced zebrafish through a mechanism involving chitinase-like protein YKL-40 analogs.. Toxicological research , vol.39, no.4 , 625-636.

Gündüz, G., Beler, M., Ünal, İ., CANSIZ, D., ALTURFAN, E. I., & KÖSE, K. N., (2023). Endotoxin of Porphyromonas gingivalis amplifies the inflammatory response in hyperglycemia-induced zebrafish through a mechanism involving chitinase-like protein YKL-40 analogs.. Toxicological research , vol.39, no.4, 625-636.

Gündüz, Gizem Et Al. "Endotoxin of Porphyromonas gingivalis amplifies the inflammatory response in hyperglycemia-induced zebrafish through a mechanism involving chitinase-like protein YKL-40 analogs.," Toxicological research , vol.39, no.4, 625-636, 2023

Gündüz, Gizem Et Al. "Endotoxin of Porphyromonas gingivalis amplifies the inflammatory response in hyperglycemia-induced zebrafish through a mechanism involving chitinase-like protein YKL-40 analogs.." Toxicological research , vol.39, no.4, pp.625-636, 2023

Gündüz, G. Et Al. (2023) . "Endotoxin of Porphyromonas gingivalis amplifies the inflammatory response in hyperglycemia-induced zebrafish through a mechanism involving chitinase-like protein YKL-40 analogs.." Toxicological research , vol.39, no.4, pp.625-636.

@article{article, author={Gizem Gündüz Et Al. }, title={Endotoxin of Porphyromonas gingivalis amplifies the inflammatory response in hyperglycemia-induced zebrafish through a mechanism involving chitinase-like protein YKL-40 analogs.}, journal={Toxicological research}, year=2023, pages={625-636} }